Search results for " Middle Cerebral Artery"

showing 10 items of 29 documents

Molecular mechanisms underlying the neuroprotective role of atrial natriuretic peptide in experimental acute ischemic stroke

2018

Abstract Along with its role in regulating blood pressure and fluid homeostasis, the natriuretic peptide system could be also part of an endogenous protective mechanism against brain damage. We aimed to assess the possibility that exogenous atrial natriuretic peptide (ANP) could protect against acute ischemic stroke, as well as the molecular mechanisms involved. Three groups of rats subjected to transient middle cerebral artery occlusion (tMCAO, intraluminal filament technique, 60 min) received intracerebroventricular vehicle, low-dose ANP (0.5 nmol) or high-dose ANP (2.5 nmol), at 30 min reperfusion. Neurofunctional condition, and brain infarct and edema volumes were measured at 24 h after…

0301 basic medicineMAPK/ERK pathwayMalePotassium ChannelsSignaling pathwaysmedicine.drug_classMAP Kinase Signaling SystemAcute ischemic strokeDown-RegulationApoptosisBrain damagePharmacologyBiochemistryNeuroprotectionBrain Ischemia03 medical and health sciencesPhosphatidylinositol 3-Kinases0302 clinical medicineEndocrinologyAtrial natriuretic peptideNatriuretic peptideMedicineAnimalsDNA CleavageRats WistarReceptorAtrial natriuretic peptideMolecular BiologyProtein kinase BPI3K/AKT/mTOR pathwayInjections Intraventricularbusiness.industryCaspase 3Natriuretic peptide receptorsBrainInfarction Middle Cerebral ArteryStroke030104 developmental biologyNeuroprotective AgentsReperfusion InjuryK+ channelsmedicine.symptombusinessProto-Oncogene Proteins c-aktReceptors Atrial Natriuretic Factor030217 neurology & neurosurgeryAtrial Natriuretic Factorhormones hormone substitutes and hormone antagonists
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Iron-loaded transferrin (Tf) is detrimental whereas iron-free Tf confers protection against brain ischemia by modifying blood Tf saturation and subse…

2018

Despite transferrin being the main circulating carrier of iron in body fluids, and iron overload conditions being known to worsen stroke outcome through reactive oxygen species (ROS)-induced damage, the contribution of blood transferrin saturation (TSAT) to stroke brain damage is unknown. The objective of this study was to obtain evidence on whether TSAT determines the impact of experimental ischemic stroke on brain damage and whether iron-free transferrin (apotransferrin, ATf)-induced reduction of TSAT is neuroprotective. We found that experimental ischemic stroke promoted an early extravasation of circulating iron-loaded transferrin (holotransferrin, HTf) to the ischemic brain parenchyma.…

0301 basic medicineU-PAGE urea-polyacrylamide gel electrophoresisMaleClinical BiochemistryExperimental strokeBiochemistryBrain IschemiaBrain ischemia0302 clinical medicineADC apparent diffusion coefficientApotransferrinDWI diffusion-weighted imagingTANDEM-1 Thrombolysis and Deferoxamine in Middle Cerebral Artery Occlusion clinical trialrHTf rat HTfrATf rat ATflcsh:QH301-705.5chemistry.chemical_classificationNeuronslcsh:R5-920ChemistryTransferrinExtravasationNS21 a medium supplement to grow neuronspDAPK-1 phosphorylated anti-death-associated protein kinase 1NeuroprotectionStrokeWB Western blotFemalemedicine.symptomlcsh:Medicine (General)Research PaperhHTf human HTfPC12 cell line derived from a pheochromocytoma of the rat adrenal medullamedicine.medical_specialtyIron OverloadBBB blood-brain barrierNMDAR N-methyl-D-aspartate receptorDCF dihydrofluoresceinIronWGA wheat germ agglutininHTf holotransferrinTransferrin receptorBrain damageTfR transferrin receptorDeferoxamineNeuroprotectionPI propidium iodide03 medical and health sciencesBrain damageCM conditioned mediumROS reactive oxygen speciesInternal medicine4-HNE 4-hydroxynonenalTf transferrinReceptors TransferrinmedicineFeRhoNoxTM-1 probe to detect Fe2+AnimalsHumansATf apotransferrinCM-H2DCFDA 5-chloromethyl-27-dichlorodihydrofluorescein diacetateMCAO middle cerebral artery occlusionDMT-1 divalent metal transporterB-27 a medium supplement to grow neuronsReactive oxygen speciesNMDA N-methyl-D-aspartateTSAT blood transferrin saturationTransferrin saturationBlood transferrin saturation (TSAT)Organic ChemistryNIR near infraredReactive oxygen species (ROS)medicine.diseasepMCAO permanent middle cerebral artery occlusionRatsPWI perfusion-weighted imaging030104 developmental biologyEndocrinologylcsh:Biology (General)TransferrinDAPK-1 anti-death-associated protein kinaseOGD oxygen/glucose deprivationTTC 235-triphenyl-tetrazolium chlorideLipid PeroxidationMCA middle cerebral arteryApoproteinsReactive Oxygen SpeciesMRI magnetic resonance imagingtMCAO transient middle cerebral artery occlusion030217 neurology & neurosurgeryhATf human ATf
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Numbers and time doubly dissociate

2011

The magnitude dimensions of number, time and space have been suggested to share some common magnitude processing, which may imply symmetric interaction among dimensions. Here we challenge these suggestions by presenting a double dissociation between two neuropsychological patients with left (JT) and right (CB) parietal lesions and selective impairment of number and time processing respectively. Both patients showed an influence of task-irrelevant number stimuli on time but not space processing. In JT otherwise preserved time processing was severely impaired in the mere presence of task-irrelevant numbers, which themselves could not be processed accurately. In CB, impaired temporal estimatio…

AdultMaleCognitive NeuroscienceIntelligenceNumbers and timeMagnitude (mathematics)Experimental and Cognitive PsychologyNeuropsychological TestsCombinatoricsExecutive FunctionBehavioral NeuroscienceDiscrimination PsychologicalHumansAttentionTime processingProblem SolvingSize PerceptionAgedIntelligence TestsSettore M-PSI/02 - Psicobiologia E Psicologia FisiologicaParietal lobeInfarction Middle Cerebral ArteryNumerosity adaptation effectMiddle AgedMagnitude processingMagnetic Resonance ImagingData Interpretation StatisticalSpace PerceptionMental RecallTime PerceptionFemaleNumerical estimationPsychologySocial psychologyPhotic StimulationPsychomotor PerformanceNeuropsychologia
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Single-dose ebselen does not afford sustained neuroprotection to rats subjected to severe focal cerebral ischemia

2004

Oxygen free radicals have been involved in the pathophysiology of cerebral ischemia, especially after spontaneous or thrombolytic reperfusion. In this study with rats, we have combined a severe focal ischemic insult (2 h) and a prolonged reperfusion time (7 days) to assess the possible sustained neuroprotective effect of ebselen (10 or 100 mg/kg), a small, lipophilic organoselenium compound which mimics glutathione peroxidase. Parietal cortical perfusion was measured by laser-Doppler flowmetry, and focal cerebral ischemia was carried out by the intraluminal thread method. We have measured plasma selenium levels, brain reduced glutathione levels, as a marker of oxidative stress, and infarct …

AzolesMaleTime FactorsCentral nervous systemDrug Evaluation PreclinicalIschemiaAdministration OralIsoindolesPharmacologymedicine.disease_causeNeuroprotectionDrug Administration ScheduleBrain IschemiaSeleniumchemistry.chemical_compoundOrganoselenium CompoundsAnimalsMedicineRats WistarBrain ChemistryPharmacologychemistry.chemical_classificationbusiness.industryEbselenCerebral infarctionGlutathione peroxidaseBody WeightBrainInfarction Middle Cerebral ArteryGlutathionemedicine.diseaseGlutathioneRatsOxidative Stressmedicine.anatomical_structurechemistrySpainAnesthesiaReperfusionReactive Oxygen SpeciesbusinessOxidative stressEuropean Journal of Pharmacology
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Mildronate treatment improves functional recovery following middle cerebral artery occlusion in rats

2011

Mildronate (3-(2,2,2-trimethylhydrazinium) propionate) is an inhibitor of l-carnitine biosynthesis and an anti-ischemic drug. In the present study, we investigated the effects of mildronate in rats following focal cerebral ischemia. Male Wistar rats were subjected to transient occlusion of the middle cerebral artery (MCAO) for 90min, followed by the intraperitoneal administration of mildronate at doses of 100 and 200mg/kg 2h after reperfusion and then daily for an additional 14days. The beam-walking, rota-rod and cylinder tests were used to assess sensorimotor function, and vibrissae-evoked forelimb-placing and limb-placing tests examined responses to tactile and proprioceptive stimulation.…

Brain InfarctionMaleIschemiaStimulationPharmacologyRotarod performance testBrain ischemiaBehavioral NeuroscienceAdjuvants ImmunologicTandem Mass SpectrometryCarnitinemedicine.arterymedicineAnimalsRats WistarStrokeChromatography High Pressure LiquidAnalysis of VarianceDose-Response Relationship Drugbusiness.industryExtremitiesInfarction Middle Cerebral ArteryRecovery of Functionmedicine.diseaseRatsBetaineDose–response relationshipRotarod Performance TestVibrissaeMiddle cerebral arterySystemic administrationbusinessNeuroscienceLocomotionPsychomotor PerformanceMethylhydrazinesBehavioural Brain Research
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Regulatory T Cells Accumulate and Proliferate in the Ischemic Hemisphere for up to 30 Days after MCAO

2012

Local and peripheral immune responses are activated after ischemic stroke. In our present study, we investigated the temporal distribution, location, induction, and function of regulatory T cells (Tregs) and the possible involvement of microglia, macrophages, and dendritic cells after middle cerebral artery occlusion (MCAO). C57BL/6J and Foxp3EGFP transgenic mice were subjected to 30 minutes MCAO. On days 7, 14, and 30 after MCAO, Tregs and antigen presenting cells were analyzed using fluorescence activated cell sorting multicolor staining and immunohistochemistry. A strong accumulation of Tregs was observed on days 14 and 30 in the ischemic hemisphere accompanied by the elevated presence …

CD4-Positive T-LymphocytesGenetically modified mousePathologymedicine.medical_specialtyTime FactorsAntigen-Presenting CellsMice Transgenicchemical and pharmacologic phenomenaLymphocyte ActivationT-Lymphocytes RegulatoryNeuroprotectionFlow cytometryMice03 medical and health sciences0302 clinical medicineImmune systemGenes ReportermedicineAnimalsLymphocyte CountIL-2 receptorAntigen-presenting cellCell Proliferation030304 developmental biologyHomeodomain Proteins0303 health sciencesmedicine.diagnostic_testMicrogliabusiness.industryInterleukin-2 Receptor alpha SubunitFOXP3Forkhead Transcription FactorsInfarction Middle Cerebral Arteryhemic and immune systemsFlow CytometryImmunohistochemistryMice Inbred C57BLmedicine.anatomical_structureNeurology030220 oncology & carcinogenesisImmunologyOriginal ArticleNeurology (clinical)CorrigendumCardiology and Cardiovascular Medicinebusiness030217 neurology & neurosurgeryJournal of Cerebral Blood Flow & Metabolism
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Subventricular Zone-Derived Neuroblasts Migrate and Differentiate into Mature Neurons in the Post-Stroke Adult Striatum

2006

Recent studies have revealed that the adult mammalian brain has the capacity to regenerate some neurons after various insults. However, the precise mechanism of insult-induced neurogenesis has not been demonstrated. In the normal brain, GFAP-expressing cells in the subventricular zone (SVZ) of the lateral ventricles include a neurogenic cell population that gives rise to olfactory bulb neurons only. Herein, we report evidence that, after a stroke, these cells are capable of producing new neurons outside the olfactory bulbs. SVZ GFAP-expressing cells labeled by a cell-type-specific viral infection method were found to generate neuroblasts that migrated toward the injured striatum after middl…

Doublecortin Domain ProteinsTime FactorsPopulationGreen Fluorescent ProteinsSubventricular zoneFluorescent Antibody TechniqueCell CountNerve Tissue ProteinsStriatumBiologyAnimals Genetically ModifiedMiceNeuroblastCell MovementNeuroblast migrationLateral VentriclesmedicineAnimalseducationNeuronseducation.field_of_studyMice Inbred ICRGeneral NeuroscienceStem CellsNeurogenesisNeuropeptidesCell DifferentiationInfarction Middle Cerebral ArteryArticlesCorpus StriatumOlfactory bulbStrokeDisease Models Animalmedicine.anatomical_structurenervous systemGanglion mother cellNeuroscienceMicrotubule-Associated Proteins
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EGFL7 ligates αvβ3 integrin to enhance vessel formation

2013

Angiogenesis, defined as blood vessel formation from a preexisting vasculature, is governed by multiple signal cascades including integrin receptors, in particular integrin αVβ3. Here we identify the endothelial cell (EC)-secreted factor epidermal growth factor-like protein 7 (EGFL7) as a novel specific ligand of integrin αVβ3, thus providing mechanistic insight into its proangiogenic actions in vitro and in vivo. Specifically, EGFL7 attaches to the extracellular matrix and by its interaction with integrin αVβ3 increases the motility of EC, which allows EC to move on a sticky underground during vessel remodeling. We provide evidence that the deregulation of EGFL7 in zebrafish embryos leads …

EGF Family of ProteinsEmbryo NonmammalianAngiogenesisAmino Acid MotifsImmunologyIntegrinGene ExpressionMice NudeEndothelial Growth FactorsBiochemistryCollagen receptorMiceCell MovementCell AdhesionHuman Umbilical Vein Endothelial CellsmedicineAnimalsHumansImmunoprecipitationPhosphorylationZebrafishbiologyReverse Transcriptase Polymerase Chain ReactionChemistryCalcium-Binding ProteinsInfarction Middle Cerebral ArteryVenous plexusCell BiologyHematologyIntegrin alphaVbeta3ImmunohistochemistryExtracellular MatrixCell biologyEndothelial stem cellHEK293 Cellsmedicine.anatomical_structureIntegrin alpha MImmunologybiology.proteinBlood VesselsRNA InterferenceIntegrin beta 6Protein BindingBlood vesselBlood
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Epidural hematoma after cochlear implantation in a 2.5-year-old boy.

2005

OBJECTIVE Report a case of an epidural hematoma after cochlear implantation in a 2.5-year-old boy, the diagnostic and therapeutical emergency management, as well as the postinterventional course and rehabilitation of the child. STUDY DESIGN Retrospective case review. PATIENT Two and a half-year-old boy, suffering from early onset, profound sensorineural hearing loss had been diagnosed at an age of 1.5 years, which had been more severe on the right side initially, but had progressed to bilateral deafness. INTERVENTION AND COMPLICATION: Cochlear implantation on the left side, followed up by an extensive epidural hematoma, causing intracranial compression with a midline shift of 15 mm to the r…

Hematoma Epidural CranialMaleReoperationmedicine.medical_specialtyCritical CareInfarctionBilateral DeafnessAudiologyDeafnessDiagnosis DifferentialInfarction Posterior Cerebral ArteryEpidural hematomaPostoperative ComplicationsMidline shiftmedicine.arteryAnterior cerebral arterymedicineElectrocoagulationHumansDominance CerebralNeurologic Examinationbusiness.industryInfarction Middle Cerebral Arterymedicine.diseaseCochlear ImplantationMagnetic Resonance ImagingMeningeal ArteriesSensory SystemsTentoriumTemporal LobeSurgeryEpistaxisOtorhinolaryngologyIntracranial EmbolismChild PreschoolNeurology (clinical)ImplantOccipital LobeComplicationbusinessTomography X-Ray ComputedFollow-Up StudiesOtologyneurotology : official publication of the American Otological Society, American Neurotology Society [and] European Academy of Otology and Neurotology
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Endogenous THBD (Thrombomodulin) Mediates Angiogenesis in the Ischemic Brain—Brief Report

2020

Objective: THBD (thrombomodulin) is part of the anticoagulant protein C-system that acts at the endothelium and is involved in anti-inflammatory and barrier-stabilizing processes. A recombinant soluble form of THBD was shown to have protective effects in different organs, but how the endogenous THBD is regulated during ischemia, particularly in the brain is not known to date. The aim of this study was to investigate the role of THBD, especially in brain endothelial cells, during ischemic stroke. Approach and Results: To induce ischemic brain damage, we occluded the middle cerebral artery of mice. We found an increased endothelial expression of Thbd in the peri-infarct area, whereas in the …

Male0301 basic medicineEndotheliumAngiogenesismedicine.drug_classThrombomodulinNeovascularization PhysiologicInflammationEndogeny030204 cardiovascular system & hematologyThrombomodulinlaw.invention03 medical and health sciences0302 clinical medicinelawAnimalsMedicineStrokeCells CulturedMice Knockoutbusiness.industryAnticoagulantBrainEndothelial CellsInfarction Middle Cerebral Arterymedicine.disease3. Good healthMice Inbred C57BLDisease Models Animal030104 developmental biologymedicine.anatomical_structureRecombinant DNACancer researchmedicine.symptomCardiology and Cardiovascular MedicinebusinessSignal TransductionArteriosclerosis, Thrombosis, and Vascular Biology
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